Iron deficiency and consequences on the body

Iron deficiency and consequences on the body
Iron deficiency and consequences on the body

- abnormal menstrual bleeding, - bleeding in some diseases, such as hemorrhoids, cancer, or regular use of. People in these situations may not be aware of blood loss. These types of bleeding are the main cause for feriprival anemia in men and women over the past - iron deficiency in the diet. This is a special risk for those with high iron needs, such as young children, adolescents and pregnant women, - low iron uptake of the body. This is not a common cause of iron deficiency but can occur in people with certain diseases as well as having a part of the stomach or small intestine removed surgically. Symptomatology of iron deficiency anemia has common parts with other anemias, as well as many characteristic phenomena.

Thus, the patient presents a red and painful tongue (especially when contacted with acidic foods), which is called. There are cracks in the mouthpieces called cheilita. Changes in appetite also appear to decrease (Sideropenic dysphagia). Food appetite is capricious, with geophagia (eating land), pagophagia (eating ice cubes) and pica (eating food that makes noise like, raw potatoes). The skin is pale, dry, harsh, devoid of elasticity and tendency to wrinkle.

it is dry, brittle, and the nails are thin, soft, sharpened, flat (called platonichie) or concave (koilonichie), without luster, with longitudinal strips. As with any other condition, before going to the actual treatment, try to determine the cause of iron deficiency and obviously to remove it, then resort to an iron-rich diet, that is to say red beef, liver . Iron supplements can be administered both orally and intravenously. It is recommended to combine the iron with citrus juices containing it. Avoid mixing with antacids that reduce the level of hydrochloric acid in the stomach.

It should be noted that iron supplements color the chair in black. These are anemias characterized by peripheral macrocytosis (large red blood cells) and the presence of megaloblasti (immature cells that will turn into macrocytes) in. It is due to vitamin B12 deficiency (cobalamin) and / or (vitamin B9). Vitamin B12 is found only in foods of animal origin (meat). Absorbs the terminal (terminal part of the small intestine) terminal, coupled with a glycoprotein (called intrinsic factor) secreted by gastric cells.

Vitamin B12 is stored in the liver, and reserves last for three to five years. Folic acid is found in vegetables and meat. It absorbs at the level of the duodenum and jejunum (the first parts of the small intestine). It is also stored in the liver, like vitamin B12, and reserves last for three to four months. Deficiency of vitamin B12 and folic acid affects the synthesis, producing an unbalance of DNA / RNA ratio, which delayes the division of the cells, hematopoiesis (producing the liver).

1. Anemia Biermer (pernicious) - occurs through the primary deficiency of vitamin B12 due to lack of intrinsic factor; 2. Parabiermeric anemia - occurs due to secondary deficiency due to other diseases: gastric neoplasm, gastric resections, chronic hepatitis, hepatic tumors;. Anemia due to folic acid deficiency. 1.

Anemia Biermer is characterized clinically by the presence of three types of syndromes (groups of signs and symptoms): a) anemic syndrome b) digestive syndrome c) neurological syndrome. a) Anemic syndrome consists of: - (headache), - jaundice (yellow pallor), - palpebral edema (eyelid infiltration) and in the calves (swollen shanks), - facies buhait, - brittle hair with tendency . b) The digestive syndrome consists of: - faulty teeth, - red tongue, depilated (without the taste buds), called Hunter glossitis, - tongue prone to contact with, - atrophy of the gastric mucosa leading to epigastric pain (in the area . c) Neurological syndrome - superficial and deep sensitivity disorders - - - paresis - psychotic disorders with paranoid states. 2.

Parabiermian anemia and folic acid deficiency anemias have the same clinical manifestations as Biermer but lack the neurological picture. The treatment of megaloblastic anemia is done by subcutaneous therapy with vitamin B12, given orally, nasally or intravenously, as appropriate. It is considered to investigate the possible presence of neoplasms, as vitamin B12 favors their production. Usually, treatment with cobalamin remains for the rest of your life. Folate therapy (folic acid salts) is particularly indicated for women who are breastfeeding in adolescents in order to prevent blood clotting disorders and.

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